Sunday, May 9, 2010

The Whole Story

I have always been (and sometimes still am) a sucker for commercials and advertisements. As a child, I was convinced that happiness came from Iron Kid’s bread. Those kids looked SO happy. I wanted to have fun like those kids. However, my mom wouldn’t let me get that bread. Did she not want me to be as happy as those children? She made me eat brown bread, with NO kids skate boarding on the package. Why, WHY, WHYYY could I not have that bread?!

I use to LOVE white rice. I would eat white rice and soy sauce at least twice a day. (It was the Asian in me). When I was about 10 years old, my family started getting brown rice. My world was turned upside down. WHY would my mother do this to me?! BROWN RICE WAS DISGUSTING. I thought I was eating rocks. I refused to eat it. One day, I decided to put my 10 year-old pride aside and give brown rice one last try. I’ve loved it ever since.

When I went home for Spring break, I cooked dinner for my grandpa, grandma, brother and sister-in-law. I put together a healthy vegan Indian meal accompanied by basmati brown rice. At the dinner table, my brother and grandpa both stated that they only cook white rice. WHITE RICE?! I sat there and short circuited for a few seconds… but I…teach people…I’m going to be a… dietitian…my own family doesn’t eat…whole grains… how has this happened?

So what’s the big deal? What’s the difference between whole grains and refined grains?

Whole grains are made up of three main parts, the endosperm, germ, and bran. The germ contains the majority of fat and the bran contains a large amount of fiber and other nutrients. The endosperm makes up about 80% of the grain and contains the bulk of carbohydrate and protein content (Slavin).

The refining process separates the bran and germ from the endosperm. When the bran gets removed, disease-preventing nutrients, phytochemicals, and antinutrients like tannins and phytic acid are also removed (Slavin). (I know ‘antinutrient’ may sound bad, but it’s not).

Whole grains obviously include the bran, germ, and endosperm, which makes food made from whole grains important sources of these nutrients and phytochemicals.

So whole grains contain dietary fiber, vitamins, minerals, and phytochemicals; fortified grains provide micronutrients such as thiamin, riboflavin, folic acid, and iron, but they are missing fiber and phytochemicals (Slavin).

What do antioxidants and phytochemicals do? So glad you asked. They reduce the damaging effects of chronic inflammation. One of these effects is DNA repair and stabilization. Now, you may not know this, but DNA is kind of important. So if your DNA is screwed, you are screwed. Antioxidants and phytochemicals also help with cell cycle control, protein repair, removal of reactive molecular species, and induce detoxification.

A study on postmenopausal women investigated the relationship of whole-grain intake with death attributed to oxidative stress and inflammation. This includes most chronic degenerative diseases, such as rheumatoid arthritis, gout, chronic obstructive pulmonary disease, emphysema, asthma, ischemia-reperfusion, ulcerative colitis, Crohns disease, type 1 and 2 diabetes, and several types of neurodegenerative diseases.

The study found a reduced risk of death in >35% of those who reported the highest intake of whole grains (Jacobs).

“Several population studies have shown correlations between increased intake of whole grain and decreased risk of developing diet-related diseases, such as cardiovascular disease (CVD), diabetes, certain cancers, and obesity (Frolich).”

To be fair, I should tell you that there was little relation between refined-grain intake and any cause of death. Refined grain consumption had a positive relationship with inflammatory death, but it was barely significant (Jacobs). So refined grains may not KILL you, but do you really want to live your life always being on the brink of death?

Another study I looked at showed that potatoes (cooked or French-fried), white rice, white bread, and carbonated beverages were most consistently associated with increased risk of type II diabetes (Barclay). (Just something to think about.) I also read that a diet high in whole grains significantly increased serum levels of retinol, a-carotene, and a-tocopherol after 16 weeks. These are vital antioxidants that everyone should want in their bodies (Jacobs)!

My suggestion for you:

If you HATE brown rice, you can try the 50/50 approach. Cook half white rice, half brown rice. Now I’ve never done this before, but brown rice takes a little longer to cook than white rice. If you are cooking the rice on the stove, you may want to add the white rice about 10 minutes after the brown rice.

If you’re feeling unusually daring and want to give up white rice completely, I suggest brown basmati rice. It has that fluffy texture that every white rice lover seems to not be able to live without.

Rice is just the beginning. There are so many different grains you can try.

Lucky you, I found a website that describes 6 different grains and how to cook them.

http://www.forecast.diabetes.org/magazine/food-thought/get-know-6-great-grains

These grains take a shorter amount of time to cook than rice! And who doesn’t love saving time? Varying your food is important; therefore, you should try to get an assortment of grains in your diet.

My personal favorite is Quinoa (KEEN-wa). It only takes about 10 minutes to cook and serves the same purpose as rice. You can roast some veggies seasoned with salt, pepper, and cayenne and place them on a bed of quinoa. Words cannot describe how delicious this is.

You also have to be careful for marketing ploys. Multi-grain might mean ‘lots of different refined grains in one bread.’ ‘Wheat bread’ is not equal to ‘whole wheat’ bread. Always read your labels. If ‘enriched wheat’ (or any other grain) is anywhere is there, put it back on the shelf.

Now you can start living a healthier life. Luckily, I had a mom who loved me so much, she didn’t care how much I whined, because she knew what was best for me and now I’m better off for it. I hope everyone has told their mom happy Mother’s Day :)

References

Barclay, A, & Petozc, P. (2008). Glycemic index, glycemic load, and chronic disease risk—a metaanalysis of observational studies. American Journal of Clinical Nutrition, 87, 627–37.

Frolich, W. (2010). Whole grain for whom and why?. Food and Nutrition Research , 54,

Jacobs, D, & Andersen, L. (2007). Whole-grain consumption is associated with a reduced risk of noncardiovascular, noncancer death attributed to inflammatory diseases in the iowa women’s health study. American Journal of Clinical Nutrition, 85, 1606-1614.

Slavin, J, & Jacobs, D. (2001). The role of whole grains in disease prevention. Journal of American Dietetic Association, 101(7),

Sunday, May 2, 2010

A big FAT lie - Part I

You hear the word FAT and instantaneously you get a negative feeling. Fat… yikes...disgusting…. filthy. The point of my blog today is to explain the health benefits from having fat in your diet. Fat does not just increase your pant size.
So the other day, I was talking to a male friend about the macronutrient composition of an optimal diet. I stated that in my opinion, an optimal diet would be composed of 65% carbs, 15% protein, and 20% fat, which is basically in sync with the USDA recommendation.
My friend replied with saying he thought his diet should be composed of only 1% fat. My jaw dropped. 1% fat?! “You sound like…” I couldn’t finish my sentence. Male pride is a sensitive thing. I thought calling him a high school girl could be damaging to his ego. As I searched for a nicer way to call him a girl, he finished my sentence.
“…a 15 year old girl?”
“Yes! EXACTLY WHAT I WAS THINKING!” I was relieved that he recognized the error in his statement. Unfortunately, many people still believe that fat is the villain.
So people think that if they don’t eat fat, they won’t be fat. WRONG. Calories are calories. Excess of calories from any source is stored as fat. Forget this low carb, high protein, low fat, carb to protein ratio BS! Excess amounts of calories from fats, carbohydrates, AND/OR protein, gets stored as fat.
Why fats get a bad rap: 1 gram of carbohydrate = 4 calories, 1 gram of protein = 4 calories, 1 gram of fat = 9 calories. So yes, this does mean you need to eat smaller amounts of fatty foods than fruits or vegetables, but you still need fat. (And I know all you protein lovers are horrified that protein and carbohydrates contain the same amount of calories; a topic for a future post).
There are different types of fat. Saturated fat (SFA), trans fat (TF), Monounsaturated Fat (MUFA), and Polyunsaturated (PUFA). In general, you want to decrease your saturated fat intake, eliminate your trans fat intake, and have your fat sources come from foods containing MUFAs and PUFAs. Within your PUFAs you have your omega 3’s and omega 6’s. These are essential fatty acids. Essential because your body does not produce them, therefore you must get them in your diet to function properly.

So maybe you’re thinking, I don’t need that much fat, because it’s just extra cushioning. Wrong. Fat is used as an energy source, cell membrane structure, lung surfactant (protects lungs from collapsing), cell to cell communication, it has structural use for glycolipids and cholesterol, acts as insulation (oh no!), it is a precursor for steroid hormones, and is a carrier of fat soluble vitamins (A,D,E, and K).
• Omega 3’s:
o Sources: canola oil, flax seeds (ground), walnuts, some eggs (read label), soy beans, fatty fish, microalgae
How does it affect my body?
• Omega-3’s are deemed as a preventer of CHD. Compared to saturated fat, trans fat, and monounsaturated fat it lowers your LDL ‘bad’ cholesterol and triglyceride level (the mechanism behind this is incredibly fascinating). They are considered healthy because they are anti-inflammatory precursors which decrease your risk for atherosclerosis.
• Omega-3s are now being associated with neurological functions and behaviors such as depression, ADHD, Alzheimer’s disease, and dementia. Studies are showing that a higher intake of omega-3s may decrease the occurrence of these dysfunctions.
• There are also studies showing that omega-3 intake may be protective against osteoporosis, but studies so far are inconclusive.
• Especially important for vegetarians: Within your omega-3 family, you have ALAs, EPAs, and DHAs. Health benefits are mostly found with EPAs and DHAs. EPAs and DHAs are not found in plants (except for seaweed, but not in a sufficient amount). Thankfully, if you eat foods containing ALAs, your body can convert it to EPA efficiently under a good environment. Meaning, low alcohol intake, a transfat free diet, omega-6 intake of no higher than 7-10%, and of course, increasing your omega-3 intake.
• Conversion from ALA to EPA to DHA is inefficient, even with high ALA intake and low intake of omega-6, trans fat, and saturated fat. Microalgae supplementation is something for vegetarians to consider.



• How do you ensure this conversion? Lowering your intake of processed foods, especially those containing trans fat, use canola oil or olive oil instead of corn oil, and eat a source of ALA every day.
Okay, so this is just what omega-3’s do. I haven’t even touched on omega-6’s or monounsaturated fat, but I will someday. Hopefully you understand that fat can be good and it is an essential part of your diet.
So, if you are eating 65% of your calories from carbohydrates, 15% from protein, and 25% from fat, you should use the following as a guideline.
A male, eating 2200 calories, you need about 550 calories from fat, if you are a woman eating 1600 calories, you need about 400 calories from fat:
2 tablespoons of peanut butter: 200 calories
¼ cup of walnuts: 164 calories
1 tablespoon of ground flaxseed: 37 calories
1 tablespoon of canola oil: 120 calories
Like every food group, you need to get a variety of different foods. You should not only eat walnuts for a fat source. You should switch it up with eating cashews or almonds.



Measurements are expressed in grams per 100 grams (3.5 oz) of food item. Trace = <0.1; (–) = 0 or no data.



References:
DeFilippis, A, & Sperling, L. (2006). Understanding omega 3's. American Heart Journal, 151(3), Retrieved from http://0-www.sciencedirect.com.catalog.llu.edu/science?_ob=ArticleURL&_udi=B6W9H-4JCBT06-8&_user=945456&_coverDate=03%2F31%2F2006&_rdoc=1&_fmt=high&_orig=search&_sort=d&_docanchor=&view=c&_acct=C000048963&_version=1&_urlVersion=0&_userid=945456&md5=863f54d2616c58c3aaf575342dc50238#secx1
Kris-Etherton, P, & Grieger, J. (2009). Dietary reference intake for dha and epa. 81(2-3), Retrieved from http://0-www.sciencedirect.com.catalog.llu.edu/science?_ob=ArticleURL&_udi=B6WPH-4WHDHR6-1&_user=945456&_coverDate=09%2F30%2F2009&_rdoc=1&_fmt=high&_orig=search&_sort=d&_docanchor=&view=c&_acct=C000048963&_version=1&_urlVersion=0&_userid=945456&md5=a78912c0a483bc4ff70c9ba767c2e023#secx9
Information was also supplied from class notes.